ABSTRACT NUMBER - 116

OPTICAL COHERENCE TOMOGRAPHY ANGIOGRAPHY OF COLLATERALS IN RETINAL VEIN OCCLUSION SHOWS VENOUS OUTFLOW ORIGIN IN THE DEEP VASCULAR COMPLEX


Belinda Leong1,2, K Bailey Freund1,2, David Sarraf3, Sean Garrity3, Kiran Vupparaboina4, Kunal Dansingani5,6

Meeting:  2018 RANZCO


SESSION INFORMATION

Date:      -

Session Title: POSTER ABSTRACT- RETINA

Session Time:      -

Purpose: To use optical coherence tomography angiography (OCTA) to evaluate the intra-retinal course of collateral vessels occurring in eyes with branch or hemispheric retinal vein occlusion (RVO) in order to demonstrate that retinal venous outflow may originate entirely in the deep vascular com- plex (DVC).

Methods: Observational retrospective review of OCTA data from eyes with branch or hemispheric RVO displaying collateral vessels within the mac- ula. Collaterals identified from fundus photography and/or fluorescein angiography not involving the perifoveal vascular ring were analyzed to determine whether they coursed through the superficial vascu- lar plexus (SVP), DVC, or both.
Results: From 23 eyes (21 branch, 1 hemispheric RVO) of 23 patients (mean age 73 11 years), 101 col- lateral vessels were identified and analyzed (mean 4.4 2.0, range: 2-9 collateral per eye). On OCTA, collat- erals appeared as curvilinear dilated flow signals con- necting veins across the horizontal raphe or on opposite sides of an occluded venous segment within the same retinal hemisphere. Of 101 collaterals ana- lyzed, nearly all showed substantially greater flow signal in the DVC, and all had some portion of their
course identified within the DVC.
Conclusions: Collateral vessels associated with branch/hemispheric RVO and identified outside the perifoveal capillary ring were all found to course through the DVC. The absence of collaterals con- fined to the SVP suggests that venous drainage does not originate in the superficial plexus. This may have important implications in understanding the pathogenesis underlying retinal vascular diseases such as paracentral acute middle maculopathy and other acute macular neuroretinopathies.

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